韓国人固有の遺伝子変異の圧倒的大部分はヒトの精神に関連していることは明白である

要 旨

1.2014年事実上はアメリカ政府食品医薬品局（ＦＤＡ）が執筆した朝鮮人を対象として分析した集団遺伝学論文の付属資料にて、地理的に近い日本人、北方系中国人が有さず、朝鮮人のみが有する固有の遺伝子変異 が生じている遺伝子リストがエクセルファイル形式で公表されています。

記事筆者が、長時間かけて、個別に手作業で調べたところ、朝鮮人固有の遺伝子変異のほぼ全てが、 何んらかの精神疾患（うつ病等）・パーソナリティー障害(間欠性爆発障害)などの 疾患感受性遺伝子であり、一言で言えば、ヒトの精神に関連する遺伝子変異でした。

2.The National Center for Biotechnology Information=アメリカ国立生物工学情報センター　ウェブサイトで朝鮮人固有の遺伝子変異が生じている遺伝子を調べると、多くの場合に関連部位として 　脳(brain)　が表示されます。従って、1の精神疾患だけではなく、脳神経変性疾患に関しても韓国人は恐らくは極めて特異であろうという結論とならざるをえません。

イントロダクション

韓国は、金大中氏がノーベル平和賞を受賞した以外にはノーベル賞受賞者ゼロであることは良く知られています。しかし、記事筆者が、2018年に調べたところ、ノーベル賞はいうまでもなく、 ガウス賞（数学）、ボルツマン賞（物理学）、ロベルト・コッホ賞(医学)等々全63もの国際的に有名な自然科学関連賞について、韓国には受賞者がただの一人もいないことを発見しました。 そのデータ詳細は、このページ最下部に表示しています
この疑いようもない客観的事実は、記事筆者をして、朝鮮人（注①）は、日本人・中国人とは異なり精神面でかなり特異な人々ではなかろうか？との疑念を生じせしめ、（注②）朝鮮人ＤＮＡに関する集団遺伝学論文を調べるきっかけとなりました。 そして、韓国人集団遺伝学者が引用したがらない下記のアメリカ政府食品医薬品局（ＦＤＡ）が執筆した論文を見つけ出しました。 英文を読める方は、methods以外の部分を是非ご自身で直接読まれることを心よりお願い申し上げます。（論文であり、機械翻訳では読めません。とにかくご自分で読まれることがなによりも重要です。それで「彼らの本質」が分かります）

Whole genome sequencing of 35 individuals provides insights into the genetic architecture of Korean population



上記ＦＤＡ論文にて、エクセル形式で朝鮮人固有の遺伝子変異が生じているリストが付属資料として公開されています

記事筆者は、上記の朝鮮人固有の遺伝子変異が生じている遺伝子について、NCBI (The National Center for Biotechnology Information )で表示されてくる論文を参照しつつ、基本的には手作業でGoogle論文検索で長時間かけて調べました。 具体的には、CSMD1（遺伝子名）＋mental、CSMD1（遺伝子名）＋GWAS、CSMD1（遺伝子名）＋cognitive等々でグーグルの論文検索をする方法です。

結 果

additional_file_7.xlsx中から朝鮮人固有の遺伝子変異の数が300以上又は特筆すべき遺伝子に限定して個々の遺伝子を手作業で調べた結果が下記の通りです。

(1)DNA中で韓国人固有の変異が生じている遺伝子の圧倒的大部分が、何らかの精神疾患、パーソナリティー障害の疾患感受性遺伝子候補でした。 ただし、記事筆者が調べた範囲では、統合失調症とうつ病については、韓国の生涯有病率は日本と明確な差はないように思えます。（うつ病については論文により数値が異なり、統合失調症に関してはほぼ確実に日本と同様です。）

また、韓国人の場合、現生人類だけが有しているNOTCH2NL遺伝子に代表されるようにヒトの頭脳全般（ヒトの精神面での進化）に関連する遺伝子について 変異が数量ベースで見れば明確に集中していました。

下記に掲げた遺伝子について、韓国人は、地理的に近い日本人・北方系中国人が有していない韓国人固有の、韓国人だけの変異を有しているのだという点をご理解ください。

(2)ただし、病理遺伝学論文は、Ａという論文で、遺伝子名〇〇〇が疾病名〇〇〇と関連がありそうだとする病理遺伝学論文が出された後、 しばらくすると、関連性は薄いという別の論文が出されるケースが多い点にはくれぐれもご留意ください。

(3)論文名は、この記事最下部に記載しています

(4)下の表内でNCBIと略称しているのは、非常に有名な The National Center for Biotechnology Information=アメリカ国立生物工学情報センター　です。

①＝遺伝子名
②＝FDA論文が抽出した韓国人に固有の遺伝子変異数(additional_file_7.xlsx中のSNV1)
③＝知的障害、精神疾患、パーソナリティー障害等の名称

なお、paper[144] の付属資料中の遺伝子リストにより、遺伝子の長さ（単位：bp）≒遺伝子の塩基数と韓国人固有の変異数（塩基数）が比例関係にないことは確認済みです
（例）PRIM2遺伝子は、長さ4,668(bp)、5033の韓国人固有の変異、MAGI2遺伝子は、長さ31,624(bp)で506の韓国人固有の変異

スマホの場合、指で右へスクロール

結論

①世界中の集団遺伝学研究者は、韓国人集団遺伝学者がほとんど引用しないため、読まれることが少く引用数も少ないが、事実上はアメリカ政府食品医薬品局（ＦＤＡ）毒物研究センターが執筆した論文を読むべきである。
②日本人の病理遺伝学研究者は、ＦＤＡ論文にて抽出された朝鮮人固有の遺伝子変異リストに基づき、研究を進めるべきである。現状では、このリストは、日本で全く知られていない。 また、精神科医は、在日韓国・朝鮮人及び帰化済み朝鮮人への投薬時には、特段の配慮が必要である
③日本の文化人類学者は、韓国社会の異様性・病身性を徹底的に分析するべきである。その際には、ＤＮＡが朝鮮人である者の協力を絶対に求めるべきではない。
④日本人の起源を探求している研究者は、ＤＮＡが朝鮮人である者を研究グループから絶対に排除するべきである。

記事筆者は、アメリカ政府食品医薬品局（ＦＤＡ）毒物研究センターが執筆した論文及び同論文にて抽出された韓国人固有の遺伝子変異リストと上記の結果からみて、韓国人は精神面から見た場合には、完全なDNA異常集団であると絶対の自信をもって言い切る。

そして、韓国人の精神面でのDNAの異常性こそ、全63もの国際的な科学関連賞の受賞者がただの一人もいないという客観的事実に完全に合致し、かつ、韓国の合計特殊出生率の異常を極めた低下（2022年で0.78）が端的に示すように病身国家韓国を生み出した主原因である


>



＊論文名一覧
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[cited from the above paper]
Here, we aimed to identify genetic overlaps at the gene level between 7 mental disorders (schizophrenia, autism spectrum disorder, major depressive disorder, anorexia nervosa, ADHD, bipolar disorder and anxiety), 8 brain morphometric traits, 2 cognitive traits (educational attainment and general cognitive function) and 9 personality traits (subjective wellbeing, depressive symptoms, neuroticism, extraversion, openness to experience, agreeableness and conscientiousness, children’s aggressive behaviour, loneliness) based on publicly available GWASs.

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[cited from the above paper]
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[cited from above paper]
According to the results of the current study, there might be a chance that variations in LSAMP gene play a role in pathoaetiology of suicidal behaviour.

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[cited from the above paper] Abstract
Relational complexity (RC) is a metric reflecting capacity limitation in relational processing. It plays a crucial role in higher cognitive processes and is an endophenotype for several disorders. However, the genetic underpinnings of complex relational processing have not been investigated. Using the classical twin model, we estimated the heritability of RC and genetic overlap with intelligence (IQ), reasoning, and working memory in a twin and sibling sample aged 1529 years (N = 787). Further, in an exploratory search for genetic loci contributing to RC, we examined associated genetic markers and genes in our Discovery sample and selected loci for replication in four independent samples (ALSPAC, LBC1936, NTR, NCNG), followed by metaanalysis (N>6500) at the single marker level. Twin modelling showed RC is highly heritable (67%), has considerable genetic overlap with IQ (59%), and is a major component of genetic covariation between reasoning and working memory (72%). At the molecular level, we found preliminary support for four singlemarker loci (one in the gene DGKB), and at a genebased level for the NPS gene, having influence on cognition. These results indicate that genetic sources influencing relational processing are a key component of the genetic architecture of broader cognitive abilities. Further, they suggest a genetic cascade, whereby genetic factors influencing capacity limitation in relational processing have a flowon effect to more complex cognitive traits, including reasoning and working memory, and ultimately, IQ.

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[cited from the above paper] Twentytwo of the DMRs identified were within the major histocompatibility complex (MHC; Fig. 3), which has been implicated in the pathogenesis of SZ through a largescale GWAS.16 In addition, we identified DMRs within two additional genes (IGSF9B, CNTN4) that showed genomewide association with SZ in the same study.
Two additional DMRs were identified within genes associated with SZ at the genomewide significant level by the SZ Working Group of the Psychiatric Genomics Consortium (PGC).16 These were within the genes IGSF9B and CNTN4, both of which function as cell adhesion molecules. Two largescale epigenomewide association studies of SZ have recently been reported.12,13 These studies reported significant differential methylation in RPTOR: a gene in which we identified a DMR. RPTOR is a key component of mTOR signalling, which has been implicated in synaptic plasticity.36

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YiSian Lin
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[cited]
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Genomewide association study of cognitive functions and educational attainment in UK Biobank (N=112 151)
G Davies et al.
Nature Published: 05 April 2016

paper[89]
Implication of LRRC4C and DPP6 in neurodevelopmental disorders
Gilles Maussion et al.
Published online 2016 Oct 19


paper[90]
A Pilot Study on EarlyOnset Schizophrenia Reveals the Implication of Wnt, Cadherin and Cholecystokinin Receptor Signaling in Its Pathophysiology
Malgorzata Marta Drozd et al.
Published online 2021
[cited from the above paper]
OR4C5 is a gene predicted by GDI to be highly damaging.

[notes]
# of SNVs in SNV1/ns is 21.
"# of SNVs in SNV35/ns" is 11.

paper[91]
Identification of genes and gene pathways associated with major depressive disorder by integrative brain analysis of rat and human prefrontal cortex transcriptomes
K Malki et al.
Nature Published: 03 March 2015


paper[92]
Evolutionary History and Genome Organization of DUF1220 Protein Domains
Michael Dickens et al.
September 2012

paper[93]
Whole Exome Sequencing in dense families suggests genetic pleiotropy amongst Mendelian and complex neuropsychiatric syndromes
Suhas Ganesh et al.
November 5, 2021

paper[94]
Differences in human and chimpanzee gene expression patterns define an evolving network of transcription factors in brain
Katja Nowick et al.
Published online 2009 Dec 10



paper[95]
An alternative splicing hypothesis for neuropathology of schizophrenia: evidence from studies on historical candidate genes and multiomics data
ChuYi Zhang et al.
Nature Published: 08 March 2021



paper[96]
Functional analysis of schizophrenia genes using GeneAnalytics program and integrated databases
Tharani Sundararajan et al.
Published online 2017 Oct 13



paper[97]
Genomewide association study of borderline personality disorder reveals genetic overlap with bipolar disorder, major depression and schizophrenia　
S H Witt et al.
Nature Published: 20 June 2017

paper[98]
Genomewide association study of antidepressant response: involvement of the inorganic cation transmembrane transporter activity pathway
Enrico Cocchi et al.
18 April 2016

paper[99]
GenomeWide DNA Methylation Changes Associated with Intermittent Explosive Disorder: A GeneBased Functional Enrichment Analysis
Janitza L MontalvoOrtiz, PhD et.al
Published: 02 November 2017

paper[100]
RBFOX1, encoding a splicing regulator, is a candidate gene for aggressivebehavior
Noèlia FernàndezCastillo et.al
2020 Jan

paper[101]
Associations between LSAMP gene polymorphisms and major depressive disorder and panic disorder
K Koido et.al
14 August 2012, nature

paper[102]
Integrative analysis of proteomewide association and transcriptomewide association study identifies candidate brain proteins associated with insomnia
Peilin Meng et.al
November 15th, 2021

paper[103]
Integrating genomewide association study with regulatory SNP annotation information identified candidate genes and pathways for schizophrenia
Xiao Liang et.al
2019 Jun 7

paper[104]
ポリシアル酸転移酵素遺伝子と精神疾患の関わり
羽根 正弥1,2，北島 健1，佐藤 ちひろ1
2017年10月25日

paper[105]
Paired involvement of humanspecific Olduvai domains and NOTCH2NL genes in human brain evolution
Ian T. Fiddes et.al
13 May 2019

paper[106]
LargeScale Cognitive GWAS MetaAnalysis Reveals TissueSpecific Neural Expression and Potential Nootropic Drug Targets
Max Lam　et.al
2017.11.028

paper[107]
Variants in the degron of AFF3 are associated with intellectual disability, mesomelic dysplasia, horseshoe kidney, and epileptic encephalopathy
Norine Voisin et.al
6 May 2021

paper[108]
The genetics of intellectual disability: advancing technology and gene editing
Muhammad Ilyas et.al
2020 Jan 16

paper[109]
Intellectual Disability and Autism Spectrum Disorders: Causal Genes and Molecular Mechanisms
Anand K. Srivastava et.al
2014 Apr 4

paper[110]
Reciprocal Relationship between Head Size, an Autism Endophenotype, and Gene Dosage at 19p13.12 Points to AKAP8 and AKAP8L
Rebecca A　et.al
June 15, 2015

paper[111]
Genetic studies in intellectualdisability and related disorders
Lisenka E. L. M. Vissers, Christian Gilissen and Joris A. Veltman
October 2015 Nature Reviews Genetics

paper[112]
The Application of Artificial Intelligence in the Genetic Study of Alzheimer’s Disease
Rohan Mishra and Bin Li
Published online 2020 Dec 1

paper[113]
Whole Exome Sequencing in Females with Autism Implicates Novel and Candidate Genes
Merlin G. Butler et.al
7 January 2015

paper[114]
GWAS metaanalysis reveals novel loci and genetic correlates for general cognitive function: a report from the COGENT consortium
J W Trampush et.al
17 January 2017

paper[115]
A genetic association study of CSMD1 and CSMD2 with cognitive function
Lavinia Athanasiu et.al
March 2017

paper[116]
The CSMD1 genomewide associated schizophrenia risk variant rs10503253 affects general cognitive ability and executive function in healthy males
Erasmia Koiliari et.al
Schizophrenia Research 154 (2014)

paper[117]
A Discovery Resource of Rare Copy Number Variations in Individuals with Autism Spectrum Disorder
Aparna Prasad et.al
01 December 2012

paper[118]
A review of intelligence GWAS hits: Their relationship to country IQ and the issue of spatial autocorrelation
Davide Piffer
22 August 2015

paper[119]
Bioinformatic Analysis Reveals Phosphodiesterase 4DInteracting Protein as a Key Frontal Cortex Dementia Switch Gene
Judith A. Potashkin et.al
2020

paper[120]
Association Between Common Variants in RBFOX1, an RNABinding Protein, and Brain Amyloidosis in Early and Preclinical Alzheimer Disease
Neha S. Raghavan, PhD et.al
2020 Jun 22

paper[121]
Behavioural and functional evidence revealing the role of RBFOX1 variation in multiple psychiatric disorders and traits
Aet O’Leary et.al
Nature 10 August 2022

paper[122]
Variants of the AggressionRelated RBFOX1 Gene in a Population Representative Birth Cohort Study: Aggressiveness, Personality, and Alcohol Use Disorder
Mariliis Vaht
24 November 2020

paper[123]
RBFOX1 and Working Memory: From Genome to Transcriptome Revealed Posttranscriptional Mechanism Separate From AttentionDeficit/Hyperactivity Disorder
Yuanxin Zhong et.al
5 September 2022

paper[124]
RBFOX1, A Splicing Regulator, is A Candidate Gene For Aggressive Behavior
Noèlia FernàndezCastillo et.al
29, Supplement 3, 2019

paper[125]
Genetic predictors of antipsychotic response to lurasidone identified in a genome wide association study and by schizophrenia risk genes
Jiang Li et.al
February 2018

paper[126]
Casecase genome wide association analysis reveals markers differentially associated with schizophrenia and bipolar disorder and implicates calcium channel genes
D Curtis et.al
2011 Aug 1.

paper[127]
PTPRD: neurobiology, genetics, and initial pharmacology of a pleiotropic contributor to brain phenotypes
George R. Uhl and Maria J Martinez
2020 Sep 1

paper[128]
Identification of a Rare Novel KMT2C Mutation That Presents with Schizophrenia in a Multiplex Family
ChiaHsiang Chen et.al
224 November 2021

paper[129]
Contribution of Multiple Inherited Variants to Autism Spectrum Disorder (ASD) in a Family with 3 Affected Siblings
Jasleen Dhaliwal et.al
8 July 2021

paper[130]
Wholeexome sequencing identifies variants associated with structural MRI markers in patients with bipolar disorders
MiRyung Han et.al
15 April 2019

paper[131]
Severe Intellectual Disability Associated with Recessive Defects in CNTNAP2 and NRXN1
C. Zweier
SEPTEMBER 08 2011

paper[132]
A Study of the Genomic Variations Associated with Autistic Spectrum Disorders in a Russian Cohort of Patients Using WholeExome Sequencing
Ekaterina A. Gibitova et.al
20 May 2022

paper[133]
Examining nonsyndromic autosomal recessive intellectual disability (NSARID) genes for an enriched association with intelligence differences
W.D. Hill et.al
Volume 54, January–February 2016

paper[134]
Investigating regions of sharedgenetic variation in attentiondefcit/hyperactivity disorderand major depressive disorder:aGWAS meta‑analysis
Victoria Powell et.al
01 April 2021(Nature)

paper[135]
Biological annotation of genetic loci associated with intelligence in a metaanalysis of 87 740 individuals
Jonathan R. I. Coleman et.al
2019 Aug 1

paper[136]
Associations between genetic loci, environment factors and mental disorders: a genomewide survival analysis using the UK Biobank data
Peilin Meng et.al
Nature 11 January 2022

paper[137]
The Netrin1/DCC guidance cue pathway as a molecular target in depression: Translational evidence
Angélica TorresBerrío et.al
2021 Oct 15

paper[137]
Genomewide Association Study of Autism Spectrum Disorder in the East Asian Populations
Xiaoxi Liu et.al
28 August 2015

paper[138]
Genetic Variants in CSMD1 Gene Are Associated with Cognitive Performance in Normal Elderly Population
Vadim Stepanov　et.al
12 December 2017

paper[139]
Genetic variation in CSMD1 affects amygdala connectivity and prosocial behavior
Bickart, KC　et.al
September 27, 2020

paper[140]
Neuropsychological effects of the CSMD1genomewide associated schizophrenia risk variantrs10503253
G. Donohoe　et.al
20 December 2012

ただし、2016年の中国人による論文では、rs10503253置換変異と統合失調症の関係を否定している。P > 0.05と論文要旨で明言
サンプルは中国人のみである
No association between the rs10503253 polymorphism in the CSMD1 gene and schizophrenia in a Han Chinese population
Yansong Liu et.al
04 July 2016

paper[141]
GenomeWide Supported Risk Variants in MIR137, CACNA1C, CSMD1, DRD2, and GRM3 Contribute to Schizophrenia Susceptibility in Pakistani Population
Ambrin Fatima et.al
2017 Sep 11
（中国人による論文では2016年にrs10503253と統合失調症の関係を否定しているが、1年後に出た上記論文では肯定している。サンプルはパキスタン人のみである。）

paper[142]
The Complement ControlRelated Genes CSMD1 and CSMD2 Associate to Schizophrenia
Bjarte Håvik et.al
1 July 2011

paper[143]
Genomewide association study of pediatric obsessivecompulsive traits: shared genetic risk between traits and disorder
Christie L. Burton et.al
02 February 2021

paper[144]
Gene Size Matters: An Analysis of Gene Length in the Human Genome
Inês Lopes　et.al
11 February 2021

paper[145]
Brain and testis: more alike thanpreviously thought?
Bárbara Matos et.al
27 April 2021

paper[146]
Somatic Signature of BrainSpecific SingleNucleotide Variations in Sporadic Alzheimer’s Disease
Antoni Parcerisas et.al
14 May 2014

paper[147]
Genomewide association metaanalysis of 78,308 individuals identifies new loci and genes influencing human intelligence
Suzanne Sniekerset.al
Nature genetics 2017 May 22

paper[148]
Genetic Relationships Between Schizophrenia, Bipolar Disorder, and Schizoaffective Disorder
Alastair G. Cardno　Michael J. Owen
3, May 2014

paper[149]
Genetic Overlap Between Attention Deficit/Hyperactivity Disorder and Autism Spectrum Disorder in SHANK2 Gene
SL Ma et.al
27 April 2021

paper[150]
The emerging role of SHANK genes in neuropsychiatric disorders
A Guilmatre et.al
20 September 2013

paper[151]
Genetic and Functional Analyses of SHANK2 Mutations Suggest a Multiple Hit Model of Autism Spectrum Disorders
CS Leblond et.al
February 9, 2012

paper[152]
Genetic association between SHANK2 polymorphisms and susceptibility to autism spectrum disorder
Y Bai et.al
22 June 2018

paper[153]
Mutations in the SHANK2 synaptic scaffolding gene in autism spectrum disorder and mental retardation
Simone Berkel et.al
Nature 16 May 2010

paper[154]
SHANK2 mutations associated with Autism Spectrum Disorder cause hyperconnectivity of human neurons
Kirill Zaslavskyet.al
Nature 2019 Mar 25

paper[155]
Genomewide association study identifies 30 Loci Associated with Bipolar Disorder
Eli A Stahl et.al
Nature genetics 2019 May 1

paper[156]
Discovery of the first genomewide significant risk loci for attentiondeficit/hyperactivity disorder
Ditte Demontis　et.al
Nature Genetics 2018 Nov 26

paper[157]
Genomewide association study of over 40,000 bipolar disorder cases provides new insights into the underlying biology
Niamh Mullins et.al
Nature genetics 2021 May 17

paper[158]
Identification of common genetic risk variants for autism spectrum disorder
Jakob Grove et.al
Nature genetics 2019 Feb 25

paper[159]
Genomewide association metaanalysis in 269,867 individuals identifies new genetic and functional links to intelligence
Jeanne E Savage et.al
Nature genetics 2018 Jun 25

paper[160]
Genomewide study of immune biomarkers in cerebrospinal fluid and serum from patients with bipolar disorder and controls
Ruyue Zhang et.al
Nature 05 February 202

paper[161]
Genomewide metaanalysis of insomnia prioritizes genes associated with metabolic and psychiatric pathways
Kyoko Watanabe et.al
Nature 14 July 2022

重要論文。23andMeの協力を得て、主にオランダの大学の研究者によるバカでかいサンプル数の分析。
筆頭著者の日本人女性は、アムステルダム自由大学所属でまだ若い。このような若い日本人研究者に一刻も早く韓国人ＤＮＡの精神面での完全な異常性に気づいて欲しい。





paper[162]
Multitrait analysis for genomewide association study of five psychiatric disorders
Yulu Wu et.al
Nature 19 July 2020

paper[163]
The genetic architecture of the human cerebral cortex　
Katrina L. Grasby　et.al
Science. 2020 Mar 20

paper[164]
Genomewide association analysis of 19,629 individuals identifies variants influencing regional brain volumes and refines their genetic coarchitecture with cognitive and mental health traits　
Bingxin Zhao et.al
Nature genetics 2019 Nov 1

paper[165]
Large-scale interaction effects reveal missing heritability in schizophrenia, bipolar disorder and posttraumatic stress disorder
H J Woo et.al
Nature 11 April 2017

paper[166]論文付属のtable4から。統合失調症の最大規模ＧＷＡＳ分析論文
Biological Insights From 108 Schizophrenia-Associated Genetic Loci
Schizophrenia Working Group of the Psychiatric Genomics Consortium
Nature. 2014 Jul 24

paper[167]
Transcriptome-wide isoform-level dysregulation in ASD, schizophrenia, and bipolar disorder
Michael J. Gandal et.al
Science. 2018 Dec 14

paper[168]
Study of 300,486 individuals identifies 148 independent genetic loci influencing general cognitive functionr
Gail Davies et.al
Nature 29 May 2018

paper[169]
A combined analysis of genetically correlated traits identifies 187 loci and a role for neurogenesis and myelination in intelligence
W. D. Hill et.al
Nature 11 January 2018

paper[170]サンプルは日本人455名
Individual risk alleles of susceptibility to schizophrenia are associated with poor clinical and social outcomes
Shinji Sakamoto et.al
Nature 17 December 2015

paper[171]
Corticolimbic DCC gene co-expression networks as predictors of impulsivity in children
Jose M. Restrepo-Lozano et.al
Nature 07 April 2022

paper[172]
Genetic association of impulsivity in young adults: a multivariate study
S Khadka et.al
Nature 30 September 2014

paper[173]
Pharmacological rescue in patient iPSC and mouse models with a rare DISC1 mutation
Nam-Shik Kim et.al
Nature 03 March 2021

paper[174]
The Netrin-1/DCC guidance system: dopamine pathway maturation and psychiatric disorders emerging in adolescence
Daniel E. Vosberg et.al
Nature 28 October 2019

paper[175]
Shining a light on CNTNAP2: complex functions to complex disorders
Pedro Rodenas-Cuadrado, Joses Ho & Sonja C Vernes
Nature 29 May 2013

paper[176]
Genome-wide association study of antisocial personality disorder
M-R Rautiainen et al.
Nature genetics 06 September 2016

paper[176]
Genome-wide association study of antisocial personality disorder
M-R Rautiainen et al.
Nature 06 September 2016

paper[177]
Human-Specific Histone Methylation Signatures at Transcription Start Sites in Prefrontal Neurons　
Hennady P. Shulha et al.
November 20, 2012

paper[178]
Genome-wide association study identifies five new schizophrenia loci　
The Schizophrenia Psychiatric Genome-Wide Association Study (GWAS) Consortium
Nature genetics 2011 Sep 18

paper[179]
Biallelic variants in CSMD1 are implicated in a neurodevelopmental disorder with intellectual disability and variable cortical malformations　
Elizabeth A. Werren et al.
Nature 30 May 2024

paper[180]
Putative complement control protein CSMD3 dysfunction impairs synaptogenesis and induces neurodevelopmental disorders
Wei Song et al.
May 2022

paper[181]
Identification of risk loci with shared effects on five major psychiatric disorders: a genome-wide analysis
Cross-Disorder Group of the Psychiatric Genomics Consortium
2013 Feb 28

paper[182]
Identification of risk loci with shared effects on five major psychiatric disorders: a genome-wide analysis
Cross-Disorder Group of the Psychiatric Genomics Consortium
2013 Feb 28






ノーベル賞だけではない！
韓国は、63の国際的な科学関連賞受賞者が全くいない


平和賞以外のノーベル賞：日本23（日本国籍のみ）　台湾1←李遠哲（台湾生まれ台湾大学卒でアメリカと中華民国の2重国籍）のみカウント
クラフォード賞（天文学と数学、地球科学、生物科学でノーベル賞の補完的賞である）：日本5
ベンジャミン・フランクリン・メダル（科学技術全般）：日本9
ショック賞（論理学・哲学」「数学」「視覚芸術」「音楽芸術」の4部門）：日本2（数学と視覚芸術）
ウルフ賞（農業、化学、数学、医学、物理学、芸術の6部門）：日本9（内訳は、芸術1化学1数学3医学3物理学1）

①数学
フィールズ賞（数学）：日本3、ベトナム　1（ゴ・バオ・チャウ）
アーベル賞（数学）：日本0
ガウス賞（数学）：日本1
コール賞 （数学）：日本4
チャーン賞（数学）：日本1
チューリング賞(計算機科学)日本：0

②物理学
ボルツマン賞（物理学）：日本2
ローレンツメダル（理論物理学）：日本ゼロ（何故、南部氏が受賞しなかった？？？）
基礎物理学ブレイクスルー賞（物理学）：日本4
国際常温核融合学会賞（常温核融合）：日本5
ジェームス・C・マックグラディ新材料賞（物理学で対象限定）：日本7
ジュリアーノ・プレパラータ・メダル（物理学で対象限定）：日本4
チャンドラセカール賞（プラズマ物理学）：日本2、台湾1
ディラック賞（理論物理学）：日本1（南部氏のみ）
ハイネマン賞（数理物理学と天体物理学）：日本3
パノフスキー賞（素粒子物理学）：日本3
ピーター・デバイ賞（物理化学）：日本0　台湾1
ブルーノ・ロッシ賞（天体物理学）：日本1
ブルーノ・ポンテコルボ賞（素粒子物理学）：日本4
ベルント・T・マティアス賞（超伝導限定物理学）：日本6　台湾2
ポアンカレ賞（数理物理学）：日本1　台湾1

③医学・生理学
アルバート・ラスカー基礎医学研究賞（基礎医学）：日本6　台湾1
ラスカー・ドゥベーキー臨床医学研究賞（臨床医学）：日本1　台湾1
ウィリアム・コーリー賞（免疫学）：日本４
ウォーレン・アルパート財団賞（医学）：日本2
ガードナー国際賞（医学）：日本12
国際ポール・ヤンセン生物医学研究賞（生命医学）：日本1
ショウ賞（賞金が100万ドル＝1億以上なので挙げました）（医学他）：日本2
パウル・エールリヒ&ルートヴィヒ・ダルムシュテッター賞（医学）：日本4
マイエンブルク賞"（癌研究のみ対象）：日本2
マスリー賞"
ラスカー・ドゥベーキー臨床医学研究賞(臨床医学)：日本1　台湾1
ローゼンスティール賞(基礎医学)：日本4
ロベルト・コッホ賞(医学)：日本7
ワイリー賞(医学)：日本2

④化学
アーサー・C・コープ賞(有機化学)：日本2　台湾1
アーネスト・ガンサー賞(天然物化学)：日本6
ウェルチ化学賞(化学)：日本1
キラリティーメダル(化学)：日本7
グレゴリー・アミノフ賞(結晶学)：日本3　台湾1
シェーレ賞(薬学)：日本1
デービーメダル(化学)：日本1
テトラヘドロン賞(化学)：日本7
ハインリッヒ・ヴィーラント賞(生化学)：日本2
ルイザ・グロス・ホロウィッツ賞(生化学)：日本1
ロジャー・アダムス賞(有機化学)：日本2

⑤生物学 エルンスト・シエーリング賞(生物学)：日本2
クラフォード賞(生物科学)：日本4
ダーウィン・メダル(生物学)：日本1
ハインリッヒ・ヴィーラント賞(生物学)：日本2
発生生物学マーチ・オブ・ダイムズ賞(生物学)：日本1

⑦その他学術関係
ヤコブ・エリクソン賞(植物学)：日本1
F.W.クラークメダル(地球科学)：日本2
アーサー・L・デイ賞(地球物理学)：日本1　 台湾1


⑧建築
RIBAゴールドメダル日本4
プリツカー賞 日本8

⑨写真
ハッセルブラッド国際写真賞 日本4

⑩文学
ノイシュタット国際文学賞日本0
フランツ・カフカ賞 日本1


1.日本（人口：1億2500万）は最低でも221以上
2.台湾（人口：2400万）も少なくとも11以上
3.韓国（人口：5200万）は完全に受賞者が全くいない＝確率上、あり得ないが現実である。